The JGA plays a complex regulatory roleA fall in glomerular blood flow/GFR can activate the JG cells to release renin which converts angiotensinogen in blood to angiotensin I and further to angiotensin IIAngiotensin II, being a powerful vasoconstrictor, increases the glomerular blood pressure and thereby GFR.Angiotensin II activates the adrenal cortex to release aldosterone.Aldosterone causes reabsorption of Na+and H2​O from the distal parts of the tubule. This leads to an increase in blood pressure and GFR.